Tafazzin Enzyme Replacement Therapy in a Mouse Model of Barth Syndrome

Tafazzin Enzyme Replacement Therapy in a Mouse Model of Barth Syndrome

Potential Therapies for Barth Syndrome: Michael T. Chin, MD, PhD, FACC, FAHAПодробнее

Potential Therapies for Barth Syndrome: Michael T. Chin, MD, PhD, FACC, FAHA

Cardiac Mitochondrial Structure and Function in Tafazzin-knockdown MiceПодробнее

Cardiac Mitochondrial Structure and Function in Tafazzin-knockdown Mice

Cardiolipin and Barth syndrome: Miriam Greenberg, PhDПодробнее

Cardiolipin and Barth syndrome: Miriam Greenberg, PhD

Impaired fatty acid metabolism in tafazzin-deficient miceПодробнее

Impaired fatty acid metabolism in tafazzin-deficient mice

Could Other Tafazzin Products Contribute to Heart and Muscle Pathology in Barth Syndrome?Подробнее

Could Other Tafazzin Products Contribute to Heart and Muscle Pathology in Barth Syndrome?

Deletion of the Cardiolipin-specific Phospholipase Rescues Growth Defects in the Yeast TafazzinПодробнее

Deletion of the Cardiolipin-specific Phospholipase Rescues Growth Defects in the Yeast Tafazzin

The mechanism of tafazzinПодробнее

The mechanism of tafazzin

Cardiomyopathy and Myocardial Noncompaction in Barth SyndromeПодробнее

Cardiomyopathy and Myocardial Noncompaction in Barth Syndrome

Endurance training in a mouse model of Barth syndrome.Подробнее

Endurance training in a mouse model of Barth syndrome.

Barth syndrome_Biological function of tafazzin_Brainstorming about future directionsПодробнее

Barth syndrome_Biological function of tafazzin_Brainstorming about future directions

Altered Triglyceride Metabolism Contributes to Low Body Weight in Tafazzin Knock-down MiceПодробнее

Altered Triglyceride Metabolism Contributes to Low Body Weight in Tafazzin Knock-down Mice

Regulation of Cardiomyopathy in Barth Syndrome by ALCAT1Подробнее

Regulation of Cardiomyopathy in Barth Syndrome by ALCAT1

Barth syndrome: Cardiolipin remodeling by ALCAT1 regulates dilated cardiomyopathyПодробнее

Barth syndrome: Cardiolipin remodeling by ALCAT1 regulates dilated cardiomyopathy

Respiratory Chain Remodeling in Cardiac Tissue of Barth Syndrome PatientsПодробнее

Respiratory Chain Remodeling in Cardiac Tissue of Barth Syndrome Patients

The preferred acyl chain donor of the yeast tafazzin.Подробнее

The preferred acyl chain donor of the yeast tafazzin.

Modeling the Mitochondrial Myopathy of Barth Syndrome using iPSC and Heart-on-chip TechnologiesПодробнее

Modeling the Mitochondrial Myopathy of Barth Syndrome using iPSC and Heart-on-chip Technologies

Targeting cardiolipin content and composition in the taz shRNA mouse model of Barth syndromeПодробнее

Targeting cardiolipin content and composition in the taz shRNA mouse model of Barth syndrome

Monolysocardiolipin Acyltransferase-1 Expression Improves Mitochondrial FunctionПодробнее

Monolysocardiolipin Acyltransferase-1 Expression Improves Mitochondrial Function